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    A Case Report of Case Report Pursuit by Medical Student

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    Medical students often seek case reports as vehicles for academic writing opportunities, conference presentation avenues, and residency/fellowship application highlights. Here we review a case where, due to unfortunate circumstances, a student made a unique diagnosis central to proper patient clinical care, wished to write up the case subsequently, but was ultimately excluded from the final work stemming from the patient case. We review the pitfalls that occurred in the process of pursuing publication of an interesting case, the educational value of pursuing case reports for students, the necessity for strong mentorship in this process, and general principles that medical students can follow regarding case report creation to avoid being burned

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    Abstracts of the 12th Indonesian Society of Interventional Cardiology Annual Meeting (ISICAM) 2020 27-29 November 202

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    No Abstrac

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    Abstracts of the 5th InaPrevent in Conjunction with the 1st InTension Summit, 25-27 June 202

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    Indonesian Society of Interventional Cardiology Annual Meeting 2021   Abstracts: Case Report

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    Abstracts of the 29th Annual Scientific Meeting of the Indonesian Heart Association (ASMIHA) 1st ASMIHA Digital Conference, 23-25 October 202

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    Whether to a greater or lesser extent, genetics often plays an important role in the development of cardiovascular diseases. Here we report a newly identified family with familial coronary artery disease (CAD) and left ventricular non-compaction (LVNC). A 52-year-old male with acute coronary syndrome, in whom LVNC had been found "incidentally", was admitted for revascularisation. From a two-dimensional echocardiogram the epicardial layer appeared to be thin and compacted, but the apical endocardial layer of the left ventricle was extremely thickened with prominent trabeculations in the endocardial layer and deep intertrabecular recesses in a channel-like structure. Family history revealed that his 47-year-old brother also had LVNC but to a lesser degree. Their mother had two false tendons in the left ventricular apex and both parents had severe CAD. Left ventricular non-compaction is "incidentally" identified in a kindred with familial CAD. The disease expressivity varies among affected individuals. Whether it is coincidental or there is a genetic link is a question that awaits further investigation. (Cardiol J 2007; 14: 407-410

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    The 5th Indonesian Intensive and Acute Cardiovascular Care Meeting   Abstracts: Case Report
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